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Characterization of cytochrome P450 3A inactivation by cannabidiol: possible involvement of cannabidiol-hydroxyquinone as a P450 inacti- vator purchase 5mg proscar visa prostate cancer mayo clinic. Suicide inactivation of rat liver cytochrome P-450 by chloramphenicol in vivo and in vitro buy proscar with a mastercard man health doctor. Identification of the heme adduct and an active site peptide modified during mechanism-based inactivation of rat liver cytochrome P450 2B1 by secobarbital order proscar 5mg free shipping prostate cancer 6 of 10. Mechanism-based inactivation of human liver cytochrome P450 2A6 by 8-methoxypsoralen buy cheap cialis professional 20 mg. Oxidative metabolism of spironolactone: evi- dence for the involvement of electrophilic thiosteroid species in drug-mediated destruction of rat hepatic cytochrome P450 cheap provera 10mg free shipping. Inhibition of mixed-function oxidations by substrates forming reduced cytochrome P-450 metabolic-intermediate complexes. Direct characterization of the selectivity of furafylline as an inhibitor of human cytochromes P450 1A1 and 1A2. Characterization of the enzymatic and non- enzymatic peroxidative degradation of iron porphyrins and cytochrome P-450 heme. Particular ability of cytochromes P450 3A to form inhibitory P450-iron-metabolite complexes upon metabolic oxidation of aminodrugs. Evaluation of atypical cytochrome P450 kinetics with two-substrate models: evidence that multiple substrates can simultaneously bind to cytochrome P450 active sites. An in vitro model for predicting in vivo inhibition of cytochrome P450 3A4 by metabolic intermediate complex formation. Differences in the inhibition of cytochromes P450 3A4 and 3A5 by metabolite-inhibitor complex-forming drugs. Cytochrome P-450 complex formation by dirithromycin and other macrolides in rat and human livers. An evaluation of potential mechanism- based inactivation of human drug metabolizing cytochromes P450 by monoamine oxidase inhibitors, including isoniazid. Diltiazem inhibition of cytochrome P-450 3A activity is due to metabolite intermediate complex formation. Prediction of cytochrome P450 3A inhibition by verapamil enantiomers and their metabolites. Mechanism-based inactivation of cytochrome P450s 1A2 and 3A4 by dihydralazine in human liver microsomes. Inactivation of cytochrome P450 3A4 by berga- mottin, a component of grapefruit juice.

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